Download Clinical Neuroimmunology: Multiple Sclerosis and Related by Patricia K. Coyle MD (auth.), Syed A. Rizvi, Patricia K. PDF

By Patricia K. Coyle MD (auth.), Syed A. Rizvi, Patricia K. Coyle (eds.)

Clinical Neuroimmunology: a number of Sclerosis and comparable issues serves as an essential source for physicians attracted to, and working with, this very advanced and evolving department of neurology. This accomplished titleprovides an advent to uncomplicated neuroimmunology and rules of immunotherapy and likewise serves as a radical advisor to immune-mediated issues of the principal and peripheral worried process, in addition to different systemic issues with an important neuroimmunologic part. The chapters are written by means of specialists of their fields and comprise the freshest clinical details. This quantity is made to simplify complicated immunological mechanisms and to target functional patient-related concerns. Clinical Neuroimmunology: a number of Sclerosis and similar problems is a useful addition to the literature and may be of vital curiosity toneurologists, citizens and fellows, internists, and normal practitioners who deal with sufferers with neurologic problems and different systemic autoimmune diseases.

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Additional info for Clinical Neuroimmunology: Multiple Sclerosis and Related Disorders

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Meinl E, Derfuss T, Krumbholz M, et al. Humoral autoimmunity in multiple sclerosis. J Neurological Sci. 2010. 1016. 131. Derfuss T, Parikh K, Velhin S, et al. Contactin 2/TAG-1-directed autoimmunity is identified in multiple sclerosis patients and mediates gray matter pathology in animals. Proc Natl Acad Sci USA. 2009;106:8302–7. 132. Semra YK, Seidi OA, Sharief MK. Heightened intrathecal release of axonal cytoskeletal proteins in multiple sclerosis is associated with progressive disease and clinical disability.

The prevalence hypothesis argues that MS comes about as a result of a pathogen that is more common in areas of high MS prevalence. Alternatively, the hygiene hypothesis states that a heavy burden of microbial or parasitic infections creates a persistent effect on the immune system early in childhood, conferring protection against MS (and other autoimmune diseases). The effect on the immune system may include a shift from pro-inflammatory helper T-cell (Th17) profile to a Th2 profile, such as that which is seen following helminthic infections and is associated with diminished inflammation in MS [4].

Neuromolecular Med. 2010;12(4):351–61. D’Souza SD, Bonetti B, Balasingman B, et al. Multiple sclerosis Fas signaling on oligodendrocyte cell death. J Exp Med. 1996;184:2361–70. 115. Franklin RJM, Ffrench-Constant C. Remyelination in the CNS: from biology to therapy. Nat Rev Neurosci. 2008;9:839–55. 116. Kuhlmann T, Miron V, Cuo Q, et al. Differentiation block of oligodendroglial progenitor cells as a cause for remyelination failure in chronic multiple sclerosis. Brain. 2008;131:1749–58. 117. Mi S, Miller RH, Tang W, et al.

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